History and exam
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MaagklachtenPublished by: NHGLast published: 2025Key diagnostic factors
common
no suspicious features of malignancy
Suspicious features include gastrointestinal (GI) bleeding, anemia, early satiety, unexplained weight loss (>10% body weight), progressive dysphagia, odynophagia, or persistent vomiting.[4][34]
If present, they suggest GI malignancy rather than gastritis.
The risk of a person <60 years old having malignancy is considered to be very low.[33]
Other diagnostic factors
common
nausea, vomiting, and loss of appetite
Nonspecific GI symptoms of gastritis include nausea, vomiting, and loss of appetite.[33]
uncommon
altered reflexes or sensory deficits
cognitive impairment
glossitis
coexisting autoimmune disease
Patients with autoimmune gastritis may have manifestations of associated autoimmune disease (e.g., thyroid disease, idiopathic adrenocortical insufficiency, vitiligo, type 1 diabetes mellitus, and hypoparathyroidism).[25]
Risk factors
strong
Helicobacter pylori infection
Acute nonerosive gastritis is most commonly due to H pylori infection.[3][4]
Chronic infection with H pylori predisposes to atrophic gastritis and autoimmune gastritis.
H pylori infection induces a severe inflammatory response with gastric mucin degradation and increased mucosal permeability that are directly cytotoxic to the gastric epithelium.[3][4]
nonsteroidal anti-inflammatory drug (NSAID) use
Up to 10% to 20% of patients taking NSAIDs report symptoms of dyspepsia, although the prevalence may range from 5% to 50%.[18][19]
Factors identified as placing patients at increased risk for NSAID-related gastrointestinal (GI) complications include prior history of a GI event (peptic ulcer, hemorrhage), age >60 years, high dosage of NSAIDs, and concurrent use of corticosteroids or anticoagulants.[22][23]
NSAIDs inhibit prostaglandin production. This in turn decreases gastric mucosal blood flow with loss of the mucosal protective barrier.[3] NSAIDs inhibit prostaglandin production.
alcohol use/toxic ingestions
Alcohol is recognized as a risk factor for erosive gastritis. Alcohol promotes depletion of sulfhydryl compounds in gastric mucosa.[3][21] Phlegmonous gastritis is also associated with recent intake of large quantities of alcohol.[27]
A rare variant, emphysematous gastritis caused by Clostridium welchii, has been associated with ingestion of corrosive agents.[28]
previous gastric surgery
critically ill patients
Critically ill patients are at risk of developing stress-induced gastrointestinal bleeding.[9] The mechanism is unclear but involves decreased mucosal blood flow and loss of the mucosal protective barrier.[3] The main risk factors associated with clinically important hemorrhage are mechanical ventilation for >48 hours and the presence of a coagulopathy.
autoimmune disease
Autoimmune disorders associated with increased risk of autoimmune gastritis include thyroid disease, idiopathic adrenocortical insufficiency, vitiligo, type 1 diabetes mellitus, and hypoparathyroidism.[24]
weak
immunocompromise
North European or Scandinavian ancestry
Recognized risk factor for autoimmune gastritis and pernicious anemia due to vitamin B₁₂ malabsorption.[10]
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