Emerging treatments

Estrogens and selective estrogen receptor modulators (SERMs)

Estrogens and selective estrogen receptor modulators (SERMs) upregulate the expression of the suppressor of cytokine signaling-2 (SOCS2) in a dose-dependent way. The SOCS2 protein impairs growth hormone (GH)-induced Janus kinase 2 phosphorylation, thus attenuating intracellular GH signaling, and, as a consequence, reduces IGF-1 production. Estrogens per se, as well as SERMs (e.g., tamoxifen, raloxifene, and clomiphene), have all been shown to decrease plasma IGF-1 levels in acromegalic patients, and normalize them in 25% to 50% of cases. It appears that this effect requires rather low biochemical activity of the disease, with pretreatment IGF-1 levels not exceeding approximately 2.5 fold of the upper limit of normal. In addition, SERMs increase plasma testosterone levels in men. Clinical benefits of estrogen/SERM therapy in acromegaly have not been assessed, and such studies would be of importance.[50][51][52]

Preoperative treatment of GH-producing somatotropinomas

The issue of preoperative treatment of GH-producing somatotropinomas with somatostatin analogs as a means of improving the efficacy of surgical intervention has been studied extensively in uncontrolled trials with variable results. A single meta-analysis concluded that in randomized, placebo-controlled trials, pretreatment with somatostatin analogs almost doubled the rate of surgical normalization of both GH and IGF-1.[53] However, these conclusions should be interpreted with caution. First, the rate of biochemical control in placebo-treated patients was exceedingly low, and the pretreatment increased it to the level customarily observed in the outcome studies reported by experienced pituitary neurosurgeons. A second meta-analysis, however, could not confirm these conclusions.[54] While initial randomized trials assessed the biochemical control rate in the short term (i.e., 3-4 months), later studies also found higher control rates short term, but failed to document durability of control long term. A potential explanation might be that there was a carry-over effect of long-acting octreotide or lanreotide used preoperatively on the postoperative GH and IGF-1 concentrations. Thus, preoperative therapy is most likely to be useful in patients who do not have access to an expert neurosurgeon and in patients with severe complications (severe pharyngeal thickness and sleep apnea, or high-output heart failure).[12][55]​ Long-term beneficial effect of somatostatin analogs preoperative treatment has not been shown.

Paltusotine

Paltusotine, an oral somatostatin receptor type 2 agonist, was effective in maintaining IGF-I values in patients with acromegaly who switched from injectable somatostatin analog in a phase 2 clinical trial.[56] A phase 3 trial is in progress.[57]​​

Subcutaneous octreotide depot

A monthly subcutaneous depot formulation of octreotide that has greater bioavailability with faster onset and stronger suppression of IGF-1 compared to long-acting intramuscular octreotide is in phase 3 clinical trials.[58][59]​​

Use of this content is subject to our disclaimer