Differentials

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Females may have had normal pubertal development or pubertal arrest.

Present with primary or secondary amenorrhea.

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High serum follicle-stimulating hormone with low estradiol, inhibin B, and anti-Mullerian hormone.

Karyotype is indicated, microarray and genetic sequencing may be indicated (requires discussion with clinical genetics).

Ovarian antibodies may be positive.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Males may have had normal pubertal development or present with pubertal arrest.

Present with decreased libido, features of testosterone deficiency, and/or infertility.

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High serum follicle-stimulating hormone with low testosterone, inhibin B, and anti-Mullerian hormone.

Karyotype is indicated, microarray and genetic sequencing may be indicated (requires discussion with clinical genetics).

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

May present with features of androgen deficiency and a lack, delay or stop of pubertal sexual maturation.

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Blood testosterone and pituitary hormone levels are low.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Pathophysiology of delayed puberty not known.

Up to half of patients have no or nonspecific symptoms.

Common symptoms include weakness, lethargy, slow speech, cold sensation, forgetfulness, constipation, and weight gain.

On exam, patients have coarse, dry skin and bradycardia.

Unless symptoms are long-standing, patients generally have some pubertal development; females report menstrual irregularities.

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Thyroid-stimulating hormone levels are elevated in primary hypothyroidism; free thyroxine levels are decreased.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Females may present with obesity, hirsutism, primary amenorrhea, or oligomenorrhea.

May have normal pubertal development but anovulatory cycles may lead to primary amenorrhea.

On exam, there may be acanthosis nigricans, hirsutism, and obesity.

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Pelvic ultrasound may reveal polycystic ovaries with variable endometrial thickness (however, the presence of polycystic ovaries are not essential for the diagnosis of polycystic ovary syndrome).

Measurement of serum androgens reveals elevated dehydroepiandrosterone sulfate (DHEAS) and testosterone.

Fasting glucose and insulin are elevated due to insulin resistance.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Normal pubertal development but cyclic pelvic pain and lack of menarche.

On exam, girls have either a perirectal mass or a bulging hymen with hematocolpos.

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Pelvic ultrasound reveals variable abnormalities such as imperforate hymen, blood within the vagina, or thickened tissue within the vagina. The uterus and ovaries are normal.

MRI pelvis may be required.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Mullerian agenesis syndrome.

Normal-onset pubertal development except menarche.

Phenotypically female external genitalia with blind vaginal pouch.

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Pelvic ultrasound reveals variable absence of Mullerian structures.

MRI pelvis may be required.

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SIGNS / SYMPTOMS

Phenotypically female with normal timing of breast development, minimal to no pubic hair growth, and no menarche.

On exam, females have an absent or blind vaginal pouch and a palpable inguinal mass (testes).

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Chromosomal analysis reveals a 46XY male in a phenotypic female.

Pelvic ultrasound reveals the presence of testes with no ovaries or uterus.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Boys present with poor virilization at puberty, although vast majority have genital ambiguity on exam.

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Testosterone: dihydrotestosterone ratio is markedly elevated.

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

May present with oligomenorrhea in females; pubertal development is normal.

Additionally, patients have central obesity with thin extremities, nuchal fat pad, moon facies, purple striae, bruisability, and hirsutism in females.

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24-hour urinary free cortisol and morning (0800 hours) serum cortisol are elevated on dexamethasone suppression test.

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